BrdU and immunocytochemical markers

From: DARZYNKIEWICZ ZBIGNIEW (DARZYNK@nymc.edu)
Date: Fri Apr 02 1999 - 14:43:15 EST


Indeed, the acid- or heat- induced denaturation of DNA to make BrdU
accessible to antiBrdU mAb is incompatible with immunocytochemical detection
of many proteins including cell surface markers. DNase tretament, on the
other hand, is hard to control and is often associated with such loss of DNA
that it is difficullt to identify the cell cycle position or DNA ploidy. The
SPIB (Strand Break Induction by Photolysis) methodology has no such
limitations. The cells are UV-illuminated to selectively photolyze DNA in
the cells that incorporated BrdU. The resulting DNA strand breaks are
labeled the same way as for detection of apoptotic cells, with directly or
indirectly labeled nucleotides, using exogenoeus terminal transferase.
Labeling of apoptotic cells may be precluded by preincubation with
dideoxynucleotide prior to photolysis, or they can be labeled with another
color fluorochrome. Cell morphology is well preserved and the method is
compatible with ethanol or formaldehyde fixation. It can be used to
differentially identify the cell cycle position or DNA ploidy (by DNA
content analysis) combined with the detection of apoptotic cells and BrdU
incorporating cells, or alternatively, for identification of BrdU
incorporating cells combined with mmunocytochemistry  (Li et al., Exp. Cell
Res., 22: 28-37, 1996; Li et al, Int. J. Oncol, 4:1157-61, 1994).
Zbigniew Darzynkiewicz



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