RNA/DNA and G0 cells

From: DARZYNKIEWICZ ZBIGNIEW (DARZYNK@nymc.edu)
Date: Fri Jul 30 1999 - 08:42:53 EST


To the  ongoing discussion on markers that can identify Go cells let me add
few remarks. The concept of Go cells, introduced over four decades ago, was
based on functional criterium, namely, it defined cells that did not
replicate DNA for the duration equivalent of two generation times. Since
then, many attempts have been made to find morphological, metabolic,
biochemical and/or molecular markers of the noncycling cells. One of such
markers is cellular RNA content. In many cell systems (e.g. mitogen
stimulated lymphocytes, fibroblasts, keratinocytes) Go cells have > 10 fold
fewer ribosomes (rRNA) compared to their cycling counterparts (G1 cells).
Because rRNA consists over 90% of total RNA, the total cellular RNA is a
reliable marker discriminating Go from G1 cells (Cytometry, 1: 98-108, 1980)
in these cell types. It makes not much sense, therefore, to use expensive
antibodies to identify Go cells (e.g. to measure Go to G1 transition during
mitogenic stimulation of lymphocytes) when for a fraction of cost and much
faster (2-3 min) cell staining with AO or Hoechst/pyronin Y provides the
same information. It should be mentioned, however, that in some tumors the
difference between Go and G1 cells in RNA content is not as great as in the
case of e.g. lymphocytes. Furthermore, it is still debatable whether tumor
cells have genuine "Go" state. During differentiation cellular RNA content
may be increased (e.g. plasma cells), or decreased (e.g. granulocytes),
depending on particular cell phenotype. In such situations cellular RNA
content may also be a marker of differentiation, but one should know whether
its increase or decrease characterizes the particular pathway of cell
differentiation. Numerous other markers of Go vs. G1 cells are used, of
which the most notable are Ki-67 and  PCNA expression. 
Because pRB phosphorylation is the critical step committing the cell to
initiate DNA replication, we have recently proposed that the status of pRB
phosphorylation should be considered as a marker discriminating Go from G1
cells (Exp. Cell Res., 239: 104-110, 1998). Antibody that specifically
reacts with underphosphorylated pRB is commercially available (e.g. from
PharMingen). It is possible, therefore, to immunocytochemically probe
phosphorylation of pRB. 
Zbigniew Darzynkiewicz



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