RE: modulation of CD4-Don't Use CD3+CD8-!!!

From: Moore, Julie (uzm5@cdc.gov)
Date: Thu Feb 04 1999 - 15:57:29 EST


Very interesting comments-is this work published?

Julie Moore, Ph.D.
Immunology Branch, Division of Parasitic Diseases
National Center for Infectious Diseases
Centers for Disease Control and Prevention
Chamblee, GA  30341
Phone: 770-488-4948
Fax: 770-488-4454
Email:  <mailto:uzm5@cdc.gov> uzm5@cdc.gov

		-----Original Message-----
		From:	Mario Roederer [mailto:roederer@stanford.edu]
		Sent:	Wednesday, February 03, 1999 12:42 PM
		To:	Cytometry Mailing List
		Subject:	Re: modulation of CD4-Don't Use CD3+CD8-!!!


		At least two suggestions for overcoming downmodulation of
CD4 included
		gating for CD3+CD8-:

		>3. Try staining for CD3+, CD8- cells. Yeah, I don't like it
much either, but
		>it works and has been used in a number of papers.

		>... stains with CD3 and CD8, then gates the CD3+, CD8-
events and assumes
		>these
		>to be the cells which would express CD4 (has they not been
overstimulated).

		Having the advantage of 10-color FACS to explore all of the
different T
		cell subsets simultaneously, I can assert definitively that
this method can
		lead to significant artefact.  CD3+CD8- T cells are
comprised of at least
		two distinct subsets:  CD4+ and CD4- T cells.  In healthy
adults, the ratio
		of these two subsets is around 6:1.  In HIV-infected adults,
especially
		advanced stages, this ratio can be much lower, for example
1:1.  Therefore,
		the CD3+CD8- gate can be substantially contaminated with
"double-negative"
		(DN) T cells.

		This is a significant problem, because DN T cells have a
very different
		functional profile from CD4 T cells (and somewhat different
from CD8 T
		cells).  A large fraction of these cells make g-IFN, few
make IL2, and none
		make IL4.  Therefore, a CD3+CD8- gate, taken to be CD4 T
cells, will
		significant OVER-estimate the gIFN production by CD4 T
cells, and somewhat
		underestimate the IL2 or IL4 production.  (Sorry, we haven't
done other
		cytokines as yet).

		Again, in healthy adults, this is not such a major problem,
although it can
		change the apparent CD4 gIFN production by 25-50% too high.
In adults with
		low CD4 counts, however, the gIFN production by CD3+CD8- T
cells is due
		principally to DN T cells, NOT to CD4 T cells.

		(Note that DN T cells are not the only contaminants to the
CD3+CD8- subset.
		Depending on the CD8 antibody being used, you may also end
up including the
		CD8-dull T cells.  These cells are not that frequent in
healthy adults,
		but, in many situations such as BMT, chemotherapy, and HIV
(among others)
		this population can become quite prevalent.  These cells
also have a very
		high proportion of gIFN producers, and very low IL2 and
nonexistent IL4
		production.)

		mr
		



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